The introduction of infectious agents results in various responses from the central nervous system (CNS). In the earliest stage of purulent bacterial brain infection, the generalized initial reaction is cerebritis. Within the background of cellular response to the infection, cerebritis evolves into a localized abscess in a predictable series of stages. Neuroimaging of these stages reflects the underlying pathophysiology of abscess formation. Variations in the brain’s reaction at different locations and similarities in the brain’s reaction to certain agents and in the appearances of aggressive neoplasms all require correlation of medical history, neuroimaging, and results of microbiologic analysis.Brain abscess may result from a traumatic brain injury, neurosurgical procedures, contiguous spread from a local source, or hematogenous spread of a systemic infection. Immunosuppressed patients, especially those patients who have received an organ transplant, are susceptible to the development of cerebral infections

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